Skip to main content

Peanuts

Peanuts

Overview

Peanuts provide niacin (B3) for NAD+ synthesis, resveratrol (polyphenol), and plant protein supporting mitochondrial function and antioxidant networks. Niacin (Vitamin B₃): Directly converted to NAD+ via salvage pathway; food sources include chicken, turkey, tuna, salmon, mushrooms, peanuts, whole grains.

Within the BRAIN Diet framework, niacin-rich foods (e.g., salmon, chicken breast, turkey, peanuts, and mushrooms) support NAD+ availability, glutathione synthesis, and mitochondrial health [1].

Key Nutritional Highlights

  • Reports on niacin Cures Systemic NAD+ Deficiency and Improves Muscle Performance in Adult-Onset Mitochondrial Myopathy [1]
  • Peanuts provide niacin (B3) for NAD+ synthesis, resveratrol (polyphenol), and plant protein supporting mitochondrial function and antioxidant networks. [1]
  • Niacin (Vitamin B₃): Directly converted to NAD+ via salvage pathway; food sources include chicken, turkey, tuna, salmon, mushrooms, peanuts, whole grains.
  • Niacin-rich foods (e.g., salmon, chicken breast, turkey, peanuts, and mushrooms) support NAD+ availability, glutathione synthesis, and mitochondrial health.

Food Context

Synergies

  • Part of diverse plant protein strategy; dietary diversity (≥30 plant foods per week) supports microbial richness and resilience
  • Pair with grains for complete amino acid profile; grain-legume complementarity improves essential amino-acid coverage

Preparation

  • Choose dry-roasted or raw over oil-roasted to preserve nutrients and avoid excess omega-6
  • Soak to reduce phytates and improve mineral bioavailability

Essential Amino Acid Profile

Peanuts provide a strong plant protein source but are not a complete protein.

Notable amino acids:

  • Lysine

Limiting amino acids:

  • Methionine and cysteine (DIAAS ~65–70)

Protein pairing strategy:

Peanuts are rich in lysine but relatively low in sulfur-containing amino acids. Combining with grains such as rice, oats, or barley helps create a more balanced essential amino acid profile.

Recipes

no recipes found

Nutrient Tables (per 100 g)

Core nutrients

NutrientAmount per 100 g% RDA per 100 g
Energy567 kcal
Protein25.8 g
Total fat49.2 g
Saturated fat6.3 g
Carbohydrates16.1 g
Sugars4.7 g
Fibre8.5 g

Key vitamins and minerals

NutrientAmount per 100 g% RDA per 100 g
Zinc3.3 mg29.7%
Magnesium168 mg40%
Phosphorus376 mg53.7%
Manganese1.9 mg84.1%
Copper1.1 mg127.1%
Folate240 µg60%
Vitamin B10.6 mg53.3%
Vitamin B312.1 mg75.4%
Vitamin E8.3 mg55.5%

Bioactive compounds

Explicitly identified compounds, including individual fatty acids, with a defensible quantity or an explicit qualitative status. Asterisks (*) identify supplementary sources below. Unquantified or trace constituents are not automatically admitted to the Substances list.

Compound / classAmount per 100 gNotes
Linoleic Acid15.6 g
Reference intakes: US Dietary Reference Intakes for adults (19–50 years; using the higher of male/female values where they differ).
Data provenance (core / micronutrient panel): USDA FoodData Central, Peanuts, all types, raw, FDC ID 172430, SR Legacy bulk (April 2018), per 100 g edible portion, last checked 2026-08-15

Substances

Substances admitted through a supported nutrition-table row. Not every table row appears here. Cards are not BRS mappings.

10 substances in this food
Cu2+

Copper

Cofactor in redox enzymes; dopamine β-hydroxylase; iron metabolism interplay

Mg2+

Magnesium

Enzymatic cofactor (>300 reactions); neurotransmitters; mitochondria; redox balance

Mn2+

Manganese

Cofactor for MnSOD (SOD2); mitochondrial antioxidant defense

PO₄³⁻

Phosphorus (Phosphate)

Structural phosphate in ATP, phosphocreatine, phospholipids, DNA/RNA, and signalling

Zn2+

Zinc

Cofactor in neurotransmission and antioxidant enzymes; dopamine modulation

References

[1] Niacin (Vitamin B₃): Directly converted to NAD+ via salvage pathway; food sources include chicken, turkey, tuna, salmon, mushrooms, peanuts, whole grains. Pirinen & Auranen 2020. Niacin Cures Systemic NAD+ Deficiency and Improves Muscle Performance in Adult-Onset Mitochondrial Myopathy