Chocolate Quinoa Crisp Clusters
Overview
A delicious cereal-to-snack hybrid that delivers satisfying crunch and steady energy. Made with whole quinoa grains, ground flaxseed, and a touch of coconut oil, these clusters offer a low glycemic profile perfect for sustained focus and energy. Enjoy them as a breakfast cereal with milk, or grab a handful for a wholesome snack anytime.
Ingredients
Core
- 2 cups cooked quinoa, cooled & surface-dried
- 2 tbsp homemade oat flour (finely blended rolled oats)
- 1 tbsp ground flaxseed
- 2–3 tbsp unsweetened cocoa or cacao powder
- ¼ tsp fine sea salt
Wet binder
- 1–2 tbsp refined coconut oil, melted (35–45 °C)
- 2–2.5 tbsp maple syrup or date syrup
- 1 tsp vanilla extract
Optional texture boosters
- 2–3 tbsp puffed quinoa, for surface coating
- ½ tsp cinnamon
- 1–2 tbsp hemp hearts (protein upgrade)
Method
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Cook & cool the quinoa: Rinse quinoa thoroughly. Cook using 2:1 water : quinoa ratio until tender. Drain well. Spread quinoa onto a tray and let it air-dry 10–20 minutes. This reduces surface moisture and preserves the grain's outer wall, which is key for cluster crunch.
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Mix the dry ingredients: In a large bowl, combine quinoa (whole, not blended), oat flour, ground flax, cocoa/cacao, and salt. Fold gently until distributed. Avoid mashing — intact grains provide microstructure and aeration.
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Add the wet binder: Melt coconut oil (35–45 °C — not smoking). Stir maple/date syrup + vanilla into the oil. Pour into the bowl and fold until coated. The coconut oil coats the surface of the clusters and crystallises as it cools, forming a thin lipid shell that reduces moisture absorption and helps maintain the cereal-style crunch.
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Form clusters: Scoop ½ tbsp (6–8 g) portions. Lightly compress into loose clusters — not spheres. Place on a parchment-lined tray with 2 cm spacing. Small clusters offer a higher surface-to-volume ratio, leading to stronger cereal-type crunch.
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Bake low + slow: 165 °C (325 °F) — 45–55 min. Rotate tray halfway. They should smell like chocolate cereal, not brownies. The aim is dehydration, not caramelisation.
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Cool fully: Rest 20–30 minutes. As the clusters cool, the lipid shell sets and the internal structure stabilises.
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Puffed quinoa finish (optional): Place puffed quinoa in a bowl. Roll or pat the cooled clusters over the surface. This preserves the identity of puffed grains and prevents them from softening during baking.
Extra Guidance
⚠️ Do not blend cooked quinoa: Blending destroys grain walls resulting in a fudge texture. You lose aeration and crunch.
Storage: Airtight container: 7–10 days room temp. Refrigerated: max crunch retention.
Serving: Dry as a snack, as cereal with milk / oat milk / coconut milk, or mixed into granola or yogurt bowls.
Nutrition
~180–220 kcal per 30g serving · 6–8g protein · high fiber · low glycemic · moderate polyphenols
Brain Health Notes
- Quinoa provides complete plant protein with all essential amino acids, supporting neurotransmitter synthesis and steady energy release.
- Flax seeds contribute ALA omega-3, soluble fiber (mucilage), and lignans, supporting gut microbiome health and SCFA production.
- Cocoa/cacao provides polyphenols (flavanols) that support cognitive function and reduce oxidative stress.
- Coconut oil's medium-chain triglycerides provide alternative energy substrate, while its crystallisation creates a protective lipid shell that preserves texture.
- The intact grain microstructure provides mechanical fracture points and aeration, creating the characteristic crunch without relying on added fats.
- Low glycemic profile from whole grains and minimal added sugars supports stable blood glucose and reduced insulin spikes.
Foods
Biological Target Matrix
Gut Microbiome
| Substance | Foods | Mechanism of Action |
|---|---|---|
| Short-Chain Fatty Acids (SCFAs) | Byproducts of fibre fermentation; support intestinal barrier integrity; regulate immune responses; promote synthesis of key neurotransmitters such as dopamine and serotonin |
Insulin Response
| Substance | Foods | Mechanism of Action |
|---|---|---|
| Cinnamaldehyde | Supports glycemic control and improves insulin sensitivity; contributes to cinnamon's glucose regulation effects | |
| Magnesium | Supports insulin sensitivity and glucose metabolism; magnesium deficiency is associated with insulin resistance; supports enzymes involved in glucose metabolism | |
| Short-Chain Fatty Acids (SCFAs) | Propionate and butyrate improve insulin sensitivity and glucose metabolism; SCFAs produced from fiber fermentation help stabilize blood glucose and reduce insulin resistance | |
| Vitamin B1 (Thiamine) | Supports glucose metabolism and insulin sensitivity through mitochondrial function |
Neurochemical Balance
| Substance | Foods | Mechanism of Action |
|---|---|---|
| Capric Triglyceride (Tridecanoin) | Ketones produced from capric triglyceride provide ATP through mitochondrial metabolism; ATP is essential for neurotransmitter synthesis, release, and reuptake, indirectly supporting neurochemical balance by ensuring adequate energy for neuronal function | |
| Caproic Triglyceride (Tricaproin) | Ketones produced from caproic triglyceride provide ATP through mitochondrial metabolism; ATP is essential for neurotransmitter synthesis, release, and reuptake, indirectly supporting neurochemical balance by ensuring adequate energy for neuronal function | |
| Caprylic Triglyceride (Trioctanoin) | Ketones produced from caprylic triglyceride provide ATP through mitochondrial metabolism; ATP is essential for neurotransmitter synthesis, release, and reuptake, indirectly supporting neurochemical balance by ensuring adequate energy for neuronal function | |
| Choline | Essential precursor for acetylcholine synthesis, supporting memory, learning, and neuroplasticity; supports membrane phospholipid biosynthesis (PC) which is critical for membrane fluidity and neurotransmitter receptor function; phospholipid methylation (PLM) alters membrane structure, facilitating faster neuronal recovery and influencing ion channel behavior in gamma oscillations linked to attention and cognition | |
| Iron | Essential cofactor for tyrosine hydroxylase, the rate-limiting enzyme in the conversion of tyrosine to dopamine; critical for catecholamine synthesis | |
| Magnesium | Broad cofactor for neurotransmitter synthesis and receptor modulation (e.g., NMDA, GABA); functions as an NMDA receptor antagonist and GABA receptor modulator; assists enzymes involved in synthesis of dopamine and serotonin | |
| MCT (Medium-Chain Triglycerides) | Ketones produced from MCTs provide ATP through mitochondrial metabolism; ATP is essential for neurotransmitter synthesis, release, and reuptake, indirectly supporting neurochemical balance by ensuring adequate energy for neuronal function | |
| Phenylalanine | Essential amino acid that converts to tyrosine and supports catecholamine synthesis (dopamine, norepinephrine); participates in LAT1 competition at the blood-brain barrier | |
| Short-Chain Fatty Acids (SCFAs) | Propionate stimulates secretion of norepinephrine and may influence dopamine regulation; SCFAs promote synthesis of dopamine and serotonin | |
| Tryptophan | Precursor for serotonin and melatonin; brain entry competes at LAT1 with other large neutral amino acids (LNAAs); carbohydrate-rich, low-protein meals raise the plasma tryptophan:LNAA ratio because insulin pushes competing LNAAs out to muscles; can feed NAD+ synthesis via the kynurenine pathway | |
| Vitamin B6 (Pyridoxine → PLP) | Cofactor for synthesis of dopamine, serotonin, GABA, and glutamate; supports rate-limiting steps in catecholamine synthesis; requires PDXK activation with magnesium and ATP support | |
| Vitamin B9 (Folate; 5-MTHF) | Supports neurotransmitter synthesis through methylation; cofactor for dopamine synthesis alongside iron, B6, and omega-3s | |
| Zinc | Important for DNA synthesis, cell division, and neurotransmitter regulation, particularly in modulating dopamine—a key neurotransmitter implicated in ADHD; acts as an allosteric modulator of the GABA receptor; supports glutamate regulation |
Oxidative Stress
| Substance | Foods | Mechanism of Action |
|---|---|---|
| ALA (Alpha-Linolenic Acid) | Essential omega-3 fatty acid; contributes to antioxidant and membrane support | |
| Linoleic Acid (LA, n-6) | Essential fatty acid; balance with omega-3s is emphasized for optimal inflammatory tone | |
| Manganese | Essential cofactor for MnSOD (SOD2), supporting detoxification of superoxide within the mitochondrial matrix | |
| Short-Chain Fatty Acids (SCFAs) | Support antioxidant activity; butyrate enhances mitochondrial function during oxidative stress | |
| Zinc | Essential mineral that serves as a cofactor for antioxidant enzymes; works synergistically with other antioxidants; heavy metals are detoxified by metallothionein (MT) metal carrier proteins that must bind with zinc and copper |