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Beetroot

Beetroot

Overview​

Beetroot is a root vegetable characterised by naturally occurring nitrate and red-violet betalain pigments, with useful folate [1]. Nitrate and pigment levels vary with cultivar, growing conditions and preparation [1].

Within a BRAIN-aligned pattern it is included as an ordinary vegetable for this combination of pigments, nitrate and folate, not as a nitrate supplement.

Food Context​

Use beetroot as a whole vegetable rather than treating it as equivalent to a concentrated nitrate product.

Preparation​

  • Preparation can change nitrate and pigment content; published values vary substantially among cultivars and products [1].
  • Roasting is useful for flavour; it does not turn beetroot into a nitrate extract.

Recipes​

2 recipes containing this food

Mitochondrial Power Bowl

A nitrate-rich, polyphenol-dense bowl combining leafy greens, beets, berries, nuts, and early harvest olive oil

Nutrient Tables (per 100 g)

Selected food and preparation: Beets, raw. per 100 g edible portion.

Core nutrients

NutrientAmount per 100 g% reference intake per 100 g
Energy43 kcal—
Protein1.6 g—
Total fat0.2 g—
Saturated fat0 g—
Carbohydrates9.6 g—
Sugars6.8 g—
Fibre2.8 g—

Key vitamins and minerals

Vitamins and minerals supplying at least 10% of an adult reference intake per 100 g, derived from the composition panel.

NutrientAmount per 100 g% reference intake per 100 g
Manganese0.3 mg14.3%
Folate109 µg27.3%

Bioactive compounds

Compound / classAmount per 100 gNotes
Betaine129 mg*—
Sources and limitations
  • Betaine: Supporting evidence and original measurement details are available in technical provenance below. [4]
Technical provenance
Source note 1

Betaine: Zeisel et al. (2003) Table 1, corrected by the erratum: beet, raw (NDB 11080), 128.63 mg betaine per 100 g, shown here as 129 mg. Canned beets in the same table are 333.69 mg per 100 g and are not this raw page. USDA Release 2 reports 130 mg per 100 g for the same raw NDB. This is not the FDC 169145 panel used for the other nutrients. No cross-food ranking is inferred. [4]

Reference intakes: US Dietary Reference Intakes for adults (19–50 years; using the higher of male/female values where they differ).
Composition source: USDA FoodData Central, Beets, raw, FDC ID 169145, per 100 g edible portion

Substances​

7 substances in this food
Chemical structure

Betaine

Occurs directly in raw beets; 129 mg per 100 g in the erratum-corrected Zeisel assay.

Fe2+

Iron

USDA FoodData Central reports iron in Beets, raw.

Source

References​

[1] Milton-Laskibar et al. (2021). Current Knowledge on Beetroot Bioactive Compounds: Role of Nitrate and Betalains in Health and Disease. Beetroot-specific review describing nitrate and betalains as major bioactive constituents and documenting substantial variation with variety and processing.

[2] Current Knowledge on Beetroot Bioactive Compounds: Role of Nitrate and Betalains in Health and Disease (2021)

[3] USDA SR Legacy April 2018

[4] Zeisel et al. (2003). Concentrations of Choline-Containing Compounds and Betaine in Common Foods. Table 1, corrected by the erratum, reports 128.63 mg betaine per 100 g of raw beets, NDB 11080, shown as 129 mg. Canned beets in that table are 333.69 mg per 100 g. USDA Release 2 reports 130 mg per 100 g for the same raw NDB. This is separate from the main nutrient panel.

Food composition → admitted dietary relationships

These links join food composition to existing PM admissions. They do not establish that a portion reproduces an intervention, delivers an intact cofactor to tissue, or improves a clinical outcome. Derived supply retains its conversion and delivery limits.

  • Iron → BRS1-FM1-PM3 · Type A / direct
    Food form & relationship limits

    Human intake-to-dopamine responsiveness and clinical benefit were not established; the direct dopamine-metabolite evidence assessed here is preclinical deficiency evidence.

    Beets, raw; raw.

  • Magnesium → BRS1-FM1-PM3 · Type A / direct
    Food form & relationship limits

    Catalytic capacity does not show that extra dietary magnesium increases regional catecholamine clearance or improves function. COMT isoforms, genotype, SAM availability and tissue homeostasis constrain interpretation; increased clearance is not universally desirable.

    Beets, raw; raw.

  • Iron → BRS1-FM1-PM4 · direct
    Food form & relationship limits

    Human intake-to-noradrenaline responsiveness and clinical benefit were not established; the cited dopamine-metabolite and developmental evidence is not a noradrenaline-dosing trial.

    Beets, raw; raw.

  • Magnesium → BRS1-FM1-PM4 · Type A / direct
    Food form & relationship limits

    Catalytic capacity does not show that extra dietary magnesium increases regional catecholamine clearance or improves function. COMT isoforms, genotype, SAM availability and tissue homeostasis constrain interpretation; increased clearance is not universally desirable.

    Beets, raw; raw.

  • Iron → BRS1-FM1-PM5 · Type A / direct
    Food form & relationship limits

    Iron is required by the hydroxylase. That is not evidence that extra dietary iron raises brain serotonin when iron status is adequate.

    Beets, raw; raw.

  • Potassium → BRS1-FM4-PM10 · Type A / direct
    Food form & relationship limits

    Established transport-ion capacity does not show that increasing dietary potassium increases brain clearance. CNS ion regulation and Na/K-pump energy supply intervene; transporter gradients are not inferred from a food quantity. No added-salt recommendation or clinical benefit.

    Beets, raw; raw.

  • Magnesium → BRS1-FM4-PM11 · Type A / direct
    Food form & relationship limits

    Neuronal ion-manipulation experiments establish physiological capacity, not oral magnesium delivery at a pharmacological concentration, treatment of excitotoxic injury or benefit above adequacy. Brain homeostasis and membrane voltage remain determinants; more NMDA inhibition is not automatically beneficial.

    Beets, raw; raw.

  • Vitamin B9 (folate) → BRS2-FM1-PM1 · Type A / derived
    Food form & relationship limits

    This conversion pathway does not show that increasing folate intake increases tissue remethylation flux. Folic acid lowers blood homocysteine, which is a circulating marker.

    Beets, raw; raw.

  • Betaine → BRS2-FM1-PM2 · Type A / direct
    Food form & relationship limits

    Supplemental betaine lowers plasma homocysteine in healthy adults, but that change does not measure tissue BHMT activity or show a clinical benefit. Human evidence is an eight-man depletion pilot, with the challenge response in four clinically depleted participants; tissue BHMT flux was not measured. Rat donor rescue and specific inhibition support the chain. This does not establish ordinary-diet limitation, response in every tissue or benefit above adequacy. Folate’s indirect constraint on BHMT remains unresolved.

    Beets, raw; raw.

  • Magnesium → BRS2-FM1-PM3 · Type A / direct
    Food form & relationship limits

    Human MAT structural and enzymological evidence establishes catalytic capacity. Ordinary food supply is distinct from measured MAT flux; no benefit from additional magnesium in replete people or universal methylation increase is asserted.

    Beets, raw; raw.

  • Potassium → BRS2-FM1-PM3 · Type A / direct
    Food form & relationship limits

    Human MAT structures establish the specific ion relationship. Physiological potassium homeostasis and tissue uptake intervene; no claim that extra dietary potassium raises SAM flux or improves methylation is established.

    Beets, raw; raw.

  • Vitamin B9 (folate) → BRS2-FM1-PM4 · Type A / direct
    Food form & relationship limits

    Folic acid can lower blood homocysteine concentration. That concentration change is not a measured change in remethylation or transsulfuration rate.

    Beets, raw; raw.

  • Magnesium → BRS2-FM2-PM6 · Type A / direct
    Food form & relationship limits

    Human GSS crystallography and cofactor annotation support capacity. The structure contains two magnesium ions; this does not establish a dietary dose, increased blood or neuronal glutathione synthesis, or benefit above adequacy. Original one-per-subunit wording is retained in history rather than used as dietary stoichiometry.

    Beets, raw; raw.

  • Manganese → BRS3-FM2-PM4 · Type A / direct
    Food form & relationship limits

    Human SOD2 requires manganese; dietary depletion experiments measured lower heart and kidney MnSOD activity in rats. This establishes a bounded enzyme-capacity dependency, not a human dose recommendation, universal tissue response, measured net ROS balance or benefit above adequate intake.

    Beets, raw; raw.

  • Iron → BRS3-FM2-PM4 · Type A / direct
    Food form & relationship limits

    Rat diet and tissue context; the kidney result is not a uniform response across organs or a human intake threshold. Human catalase haem identity supports capacity, not benefit from extra iron. Iron can also promote non-enzymatic oxidation; higher catalase activity is not a clinical benefit.

    Beets, raw; raw.

  • Iron → BRS4-FM1-PM1 · Type A / direct
    Food form & relationship limits

    This identifies iron centres in the chain. It does not show that eating more iron, once iron status is adequate, raises electron transport.

    Beets, raw; raw.

  • Manganese → BRS4-FM2-PM5 · Type A / direct
    Food form & relationship limits

    Capacity is supported by protein biochemistry and mouse dietary manipulation; no general-population limitation, extra-intake benefit or human supplement dose is established.

    Beets, raw; raw.

  • Magnesium → BRS6-FM1-PM3 · Type A / direct
    Food form & relationship limits

    Dietary-depletion evidence is preclinical; extra intake in magnesium-replete humans is not shown here.

    Beets, raw; raw.