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Turmeric Lentil Dahl

Turmeric lentil dahl in a bowl

Overview

This warming, anti-inflammatory dahl combines turmeric's powerful curcumin with ginger's anti-inflammatory properties, high-fibre lentils for gut health, and folate-rich spinach. The combination provides strong NF-κB inhibition to reduce neuroinflammation, supports SCFA production through prebiotic fibre, and delivers essential nutrients for methylation pathways, making it an ideal evening meal for stable glucose and calmer energy.

Ingredients

  • 1 cup red lentils
  • 1 tbsp grated fresh turmeric (or 1 tsp ground turmeric)
  • 1 tsp grated ginger
  • 1 tsp cumin
  • 1 tbsp coconut oil
  • 2 cups water or stock
  • 1 cup spinach
  • Salt & pepper to taste
  • Optional: lemon juice, coriander

Method

  1. Rinse lentils thoroughly until the water runs clear.
  2. Heat coconut oil in a pot over medium heat.
  3. Add turmeric, ginger, and cumin; sauté for 1 minute until fragrant.
  4. Add lentils and water (or stock).
  5. Bring to a boil, then reduce heat and simmer for 15–20 minutes until soft and creamy.
  6. Stir in spinach and cook until wilted.
  7. Season with salt, pepper, and optional lemon juice.
  8. Serve warm.

Nutrition

~400 kcal · 18–20 g protein (≈ 40% RDA) · 13–15 g fibre (≈ 45–50% RDA) · 25–30% RDA magnesium · 40–50% RDA folate · high polyphenols

Brain Health Notes

  • Turmeric (curcumin) provides strong NF-κB inhibition, reducing neuroinflammation.
  • Ginger offers anti-inflammatory benefits and supports gut motility.
  • Lentils provide high fibre for SCFA production and microbiome health.
  • Spinach supplies folate for methylation pathways.
  • Slow, even carbohydrate release supports stable glucose and calmer evening energy.

Foods

7 foods in this recipe

Cilantro

Herb with potential heavy metal chelation properties

Ginger

Gut-brain axis support and anti-inflammatory; prokinetic for SIBO

Biological Target Matrix

Gut Microbiome

SubstanceFoodsMechanism of Action
Supported through recipe ingredients

Inflammation

SubstanceFoodsMechanism of Action
CopperParticipates in redox enzymes and antioxidant networks
Curcumin (Turmeric)Anti-inflammatory and neuroprotective effects; supports BDNF expression through polyphenol synergy
LuteinAnti-inflammatory properties; supports immune regulation
Vitamin C (Ascorbate)Antioxidant properties; supports anti-inflammatory effects
ZeaxanthinAnti-inflammatory properties; supports immune regulation
ZincSupports immune signaling; gut barrier integrity disrupted by nutrient deficiencies including zinc
β-CaroteneAnti-inflammatory properties; supports immune regulation

Methylation

SubstanceFoodsMechanism of Action
MethionineEssential amino acid that forms S-adenosylmethionine (SAMe), the universal methyl donor for neurotransmitter synthesis and membrane phospholipid methylation
Vitamin B2 (Riboflavin)FAD acts as a critical cofactor for MTHFR, linking riboflavin to homocysteine recycling and methylation capacity
Vitamin B6 (Pyridoxine → PLP)Essential cofactor in remethylation of homocysteine to methionine, which is converted to S-adenosylmethionine (SAMe); works with B2, folate, and B12
Vitamin B9 (Folate; 5-MTHF)Essential cofactor in remethylation of homocysteine to methionine, which is converted to S-adenosylmethionine (SAMe); SAMe fuels synthesis of dopamine, norepinephrine, and serotonin and drives phospholipid methylation in neuronal membranes
ZincDeficiencies in vitamins and minerals essential for methylation, such as folate, vitamin B12, and zinc, are correlated to ADHD symptoms; supplementing these micronutrients has shown potential in supporting methylation and reducing symptom severity

Neurochemical Balance

SubstanceFoodsMechanism of Action
CalciumEssential for nerve impulse transmission and neurotransmission
Capric Triglyceride (Tridecanoin)Ketones produced from capric triglyceride provide ATP through mitochondrial metabolism; ATP is essential for neurotransmitter synthesis, release, and reuptake, indirectly supporting neurochemical balance by ensuring adequate energy for neuronal function
Caproic Triglyceride (Tricaproin)Ketones produced from caproic triglyceride provide ATP through mitochondrial metabolism; ATP is essential for neurotransmitter synthesis, release, and reuptake, indirectly supporting neurochemical balance by ensuring adequate energy for neuronal function
Caprylic Triglyceride (Trioctanoin)Ketones produced from caprylic triglyceride provide ATP through mitochondrial metabolism; ATP is essential for neurotransmitter synthesis, release, and reuptake, indirectly supporting neurochemical balance by ensuring adequate energy for neuronal function
CopperCofactor in dopamine β-hydroxylase, supporting catecholamine synthesis; supports norepinephrine synthesis
IronEssential cofactor for tyrosine hydroxylase, the rate-limiting enzyme in the conversion of tyrosine to dopamine; critical for catecholamine synthesis
MagnesiumBroad cofactor for neurotransmitter synthesis and receptor modulation (e.g., NMDA, GABA); functions as an NMDA receptor antagonist and GABA receptor modulator; assists enzymes involved in synthesis of dopamine and serotonin
MCT (Medium-Chain Triglycerides)Ketones produced from MCTs provide ATP through mitochondrial metabolism; ATP is essential for neurotransmitter synthesis, release, and reuptake, indirectly supporting neurochemical balance by ensuring adequate energy for neuronal function
PhenylalanineEssential amino acid that converts to tyrosine and supports catecholamine synthesis (dopamine, norepinephrine); participates in LAT1 competition at the blood-brain barrier
PotassiumCritical for membrane potential, nerve signaling, and neuronal excitability; adequate intake balances sodium effects
TryptophanPrecursor for serotonin and melatonin; brain entry competes at LAT1 with other large neutral amino acids (LNAAs); carbohydrate-rich, low-protein meals raise the plasma tryptophan:LNAA ratio because insulin pushes competing LNAAs out to muscles; can feed NAD+ synthesis via the kynurenine pathway
TyrosineCatecholamine precursor (dopamine, norepinephrine); brain transport via LAT1 competes with other LNAAs; iron is an essential cofactor for tyrosine hydroxylase, the rate-limiting enzyme in conversion of tyrosine to dopamine; cofactors include iron, B6, folate, omega-3s, and BH₄ (tetrahydrobiopterin) to support rate-limiting steps in catecholamine synthesis
Vitamin B6 (Pyridoxine → PLP)Cofactor for synthesis of dopamine, serotonin, GABA, and glutamate; supports rate-limiting steps in catecholamine synthesis; requires PDXK activation with magnesium and ATP support
Vitamin B9 (Folate; 5-MTHF)Supports neurotransmitter synthesis through methylation; cofactor for dopamine synthesis alongside iron, B6, and omega-3s
Vitamin C (Ascorbate)Supports norepinephrine synthesis; transported in brain via SVCT2
ZincImportant for DNA synthesis, cell division, and neurotransmitter regulation, particularly in modulating dopamine—a key neurotransmitter implicated in ADHD; acts as an allosteric modulator of the GABA receptor; supports glutamate regulation

Oxidative Stress

SubstanceFoodsMechanism of Action
Coenzyme Q10 (CoQ10)Part of antioxidant network; works synergistically with vitamin E, vitamin C, lipoic acid, and glutathione
CopperIncluded in antioxidant enzyme networks; interacts with iron metabolism affecting oxidative stress
Curcumin (Turmeric)Antioxidant properties; supports antioxidant defenses
LuteinAntioxidant properties; scavenges reactive oxygen species and stabilizes cell membranes
ManganeseEssential cofactor for MnSOD (SOD2), supporting detoxification of superoxide within the mitochondrial matrix
Vitamin A (Retinoids; β-Carotene precursor)Provitamin A carotenoids (β-carotene) act as antioxidants in neural tissue; contribute to antioxidant network
Vitamin C (Ascorbate)Key water-soluble antioxidant; works within antioxidant network with vitamin E, CoQ10, and polyphenols
ZeaxanthinAntioxidant properties; scavenges reactive oxygen species and stabilizes cell membranes
ZincEssential mineral that serves as a cofactor for antioxidant enzymes; works synergistically with other antioxidants; heavy metals are detoxified by metallothionein (MT) metal carrier proteins that must bind with zinc and copper
β-CaroteneAntioxidant properties; scavenges reactive oxygen species and stabilizes cell membranes