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Arabinan

Overview​

Arabinan is a branched plant polysaccharide. Its backbone is (1→5)-linked α-L-arabinofuranose, with arabinofuranose side chains at O-2, O-3, or both. In the cell wall it is a neutral side chain of pectin, attached through rhamnose. It is not arabinogalactan, not free arabinose, and not pectin itself. This page uses arabinan for that glycan class. Sugar beet, seeds, and roots are common plant sources, but a food that contains them is not the exposure studied here.

In mice colonised with defined human-derived communities, arabinan in one pea-fibre preparation supplied a glycan whose use depended on microbial utilisation machinery and on competition with other species [1]. The food matrix and the competitors affected access. That result does not set a human intake, an optimal dose, or a host benefit, and it does not transfer to sugar-beet arabinan or to pectin in general [1].

Dietary Origin​

Direct occurrence is arabinan in plant cell walls, as a side chain of pectin. The studied exposure is narrower: arabinan in one pea-fibre preparation, given in a defined-community mouse model [1]. A linear, enzymatically debranched arabinan is a different preparation. A food that contains peas, sugar beet, or pectin is not identified as the studied fraction. No food page has a measured arabinan amount, so none is linked.

Research Spotlights & Evidence Checks​

Recipes​

no recipes found (no foods contain this substance)

Foods​

no foods found

Biological Regulatory Systems​

Biological Regulatory SystemEvidence-qualified relationshipEvidence
Gut-Brain Axis & Enteric Nervous System (BRS5)Pea-fibre arabinan is a glycan substrate in microbial competitive selection. Use depends on microbial machinery and competition.[1]

References​

[1] Patnode et al. (2019). Interspecies Competition Impacts Targeted Manipulation of Human Gut Bacteria by Fiber-Derived Glycans. Arabinan in the studied pea fibre was a glycan resource whose utilisation depended on microbial machinery and competition in defined human-derived communities in mice. No universal human response, optimal dose, or host benefit is established.