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BRS6(PM1) - Glycaemic Excursion Control

Regulation of post-prandial glucose rise and fall to reduce acute metabolic volatility and compensatory stress signalling.

Biological Role

↓ post-prandial glucose spike; ↓ glucose crash risk; ↑ stable energy availability

Dependencies (System Requirements)

KCs

Optional BRSX Modifiers

  • None listed

Cofactors (Chemical Enablement)

  • magnesium
  • chromium
  • B vitamins

Inputs (Dietary and Environmental)

oats/legumes/sweet potato → slow carbohydrates + fibre; protein/fat matrix → slower glucose appearance; vinegar / acidic foods → reduced post-prandial glucose response; refined sugar → antagonistic input

Outputs / Biological Effects

↓ post-prandial glucose spike; ↓ glucose crash risk; ↑ stable energy availability

Functional Mechanism Ownership

  • BRS6(FM1)

Intervention Dominance

  • Diet-Dominant

Constraints and Failure Modes

[INSERT_FROM_COLUMN_N_OR_SHEET_NOTES_IF_AVAILABLE]

Notes

  • Evidence Type (Column H): Human + mechanistic
  • Evidence/Notes (Column N): Core diet-dominant PM linking meal construction to cognitive energy stability.

Mechanism Summary Table

FieldValue
PM IDBRS6(PM1)
FM Ownership (Column P)BRS6(FM1)
Dose Target / Requirement (Column J)Low-to-moderate glycaemic load with fibre/protein/fat buffering
Coverage Timing (Column K)Meal
Response Type (Column L)Immediate–Hours
Functional Latency (Column M)Same meal

References

Missing Entities

  • None flagged from this row-level pass